The COVID spike protein has 4 primary pathogenic mechanisms (discovery is ongoing), regardless if it originated from a natural COVID infection or was generated by a vaccine.
- It binds to healthy cells and opens up the pathway for the COVID virion to enter into the healthy cell. It is extremely efficient at doing this causing an unusually rapid replication and increased viral load. It also sheds its remaining spike proteins creating systemic inflammation throughout the patient’s body.
- It is in and of itself highly inflammatory, especially systemic inflammation of vascular endothelium. This is further aggravated by immune monocyte cells that ingest the spike proteins and carry them throughout the patient’s body (see Dr. Bruce Patterson’s pioneering research in this area). As you will see, this inflammation often causes ischemic damage to organs resulting in the symptoms of COVID.
- 3.The spike protein is also a prion disrupting heme complexes such as hemoglobin in RBCs and cytochrome complexes in mitochondria.
- Spike protein binds to blood coagulation factor fibrinogen and induces structurally abnormal blood clots with heightened pro-inflammatory activity disrupting blood flow and perfusion to organs throughout the patient’s body (see dialyzer lab analysis wherein clots are identified but spike proteins can not be individually identified).
REMEMBER: COVID is documented to produce as many as 200 symptoms caused by mitochondrial dysfunction and by systemic vascular inflammation and abnormal blood clotting that can induce ischemic damage to virtually every organ system in our bodies.